Professor Keith Kendrick graduated in Psychology at the University of Durham in the UK in 1976 where he also did his PhD which established how sex steroid hormones can directly alter the way brain networks respond to sexually arousing stimuli. After 
positions in the Institute of Zoology in London and the University of Cambridge studying the control of affiliative behaviours he then moved to the Babraham Institute in Cambridge in 1985 where was Head of the Cognitive and Systems Neuroscience Laboratory and worked on brain mechanisms of social recognition and learning as well as affiliative bonds. His research established the central role of brain oxytocin in promoting parental behaviour and social bonds and how neural networks mediate face and odour recognition. He was made a Fellow of the Society of Biology in 1996 and from 2002-2006 he was Gresham Professor of Physic in London giving public lectures on the biomedical sciences in London. He was made an Emeritus Professor of Gresham College in 2006. He moved to his current position as full Professor in the School of Life Sciences and Technology at UESTC in September 2011 as a recipient of a 1000 Talent award and has set up a Social Cognition and Affective Neuroscience Group within the Key Laboratory of Neuroinformation. His research work at UESTC will mainly focus on establishing the social and cognitive effects of prosocial peptides in humans using behavioural, brain imaging and recording and pharmacogenetic approaches. Another associated area of research will be to establish how of social feedback can be used to promote e-learning. Additionally, he will be involved in Clinical studies investigating potential therapeutic effects of prosocial peptides for Autism and other psychiatric disorders. He will also investigate functional connectivity changes in the brains of patients with psychiatric disorders.
Recent Publications
Ge T, Kendrick KM, Feng J, (2009) A Novel Extended Granger Causal Model Approach Demonstrates Brain Hemispheric Differences during Face Recognition Learning. PLoS Computational Biology 5(11): e1000570. doi:10.1371/journal.pcbi.1000570. (IF 5.76)
Burne THJ, Johnstone ANB, Wilkinson LS, Kendrick KM. (2010) Effects of anesthetic agents on socially transmitted olfactory memories in mice. Neurobiology of Learning and Memory 93: 268-274. (IF 3.7)
Hurlemann R, Walter H, Rehme AK, Kukolja J, Santoro SC, Schmidt C, Schnell K, Musshoff F, Keysers C, Maier W, Kendrick KM, Onur OA (2010) Human amygdala reactivity is diminished by the beta-noradrenergic antagonist propranolol. Psychological Medicine 27: 1-10. (IF 5.2)
Onur OA, Schlaepfer TE, Kukolja J, Bauer A, Jeung H, Patin A, Otte D-M, Shah NJ, Maier W, Kendrick KM, Fink GR, Hurlemann R. (2010) The NMDA receptor c-agonist D-cycloserine facilitates declarative learning and hippocampal activity in humans. Biological Psychiatry doi:10.1016/j.biopsych.2010.01.022. (IF 8.67)
Hurlemann R, Patin A, Oezguer P, Onur OA, Cohen MX, Baumgartner T, Metzler S, Dziobek I, Gallinat J, Wagner M, Maier W, Kendrick KM (2010) Oxytocin enhances amygdala-dependent, socially reinforced learning and emotional empathy in humans. Journal of Neuroscience 30(14):4999 –5007. (IF 7.27)
Mihov Y, Mayer S, Musshoff F, Maier W, Kendrick KM, Hurlemann R. (2010) Socially reinforced learning in humans is beta-noradrenergic-dependent. Neuropsychologia 48:3168-3172. (IF 3.95)
Sanchez-Andrade G, Kendrick KM.(2011) Roles of α and β estrogen receptors in mouse social recognition memory: effects of gender and the estrous cycle. Hormones and Behavior 59:114-122. (IF 3.99)
Kendrick KM, Zhan Y, Fischer H, Nicol A, Zhang X, Feng J (2011) Learning alters theta amplitude, theta-gamma coupling and neuronal synchronization in inferotemporal cortex. BMC Neuroscience 12:55. (IF 3.09)
Tao H*, Guo S*, Ge T*, Kendrick KM*, Xue Z, Liu Z, Feng J. (2011) Depression uncouples brain hate circuit. Molecular Psychiatry doi:10.1038/mp.2011.127*Joint 1st authors (IF 15.47)
Striepens N, Kendrick KM*, Maier W, Hurlemann* R (2011) Prosocial effects of oxytocin and clinical evidence for its therapeutic potential. Frontiers in Neuroendocrinology doi:10.1016/j.yfrne.2011.07.001 *Joint corresponding author (IF 12.75)